Open Access

Erratum to: Inflammation, glucose, and vascular cell damage: the role of the pentose phosphate pathway

  • Concepción Peiró1,
  • Tania Romacho1, 4,
  • Verónica Azcutia1, 5,
  • Laura Villalobos1,
  • Emilio Fernández2,
  • Juan P. Bolaños2,
  • Salvador Moncada3, 6Email author and
  • Carlos F. Sánchez-Ferrer1Email author
Contributed equally
Cardiovascular Diabetology201716:25

https://doi.org/10.1186/s12933-017-0502-1

Published: 16 February 2017

The original article was published in Cardiovascular Diabetology 2016 15:82

Erratum to: Cardiovasc Diabetol (2016) 15:82 DOI 10.1186/s12933-016-0397-2

After publication of the original article [1], it became apparent that an error affecting Fig. 6 occurred during production. In the published article, Fig. 6a is missing the Western blot corresponding to G6PD.
Fig. 6

G6PD siRNA abrogates the glucose potentiation of IL1β-evoked pro-inflammatory response. a G6PD levels, determined by Western blot, in cells treated with scrambled siRNA or G6PD siRNA and submitted for 18 h IL1β (10 ng/mL) in medium containing 5.5 or 22 mmol/L glucose. b NADPH oxidase activity, determined by lucigenin-derived chemiluminescence in cells treated with scrambled siRNA and G6PD siRNA and exposed to IL1β (10 ng/mL) during 18 h of incubation, in medium initially containing 5.5 or 22 mmol/L glucose. Results are the mean ± standard error of 3–6 separate experiments expressed as percentage of the relative light units produced by 10 ng/mL IL1β in control cells incubated in a medium initially containing 5.5 mmol/L glucose (380.1 ± 59.7 RLU/µg protein min−1). c, d iNOS levels, determined by Western blot, in cells untreated or treated with scrambled (scr)-siRNA and G6PD siRNA and exposed to IL1β (10 ng/mL) during 18 h of incubation in medium initially containing 5.5 or 22 mmol/L glucose. The gels and blots are representative of 3–5 separate experiments, while the bars are expressed as percentage of the activation or the expression produced by treatment with IL1β in sc-siRNA cells incubated in a medium with 5.5 mmol/L glucose. *P < 0.05 vs respective control (c). P < 0.05 vs respective value in 5.5 mmol/L glucose. # P < 0.05 vs respective value in scr-siRNA

The correct version of the figure was submitted by the author, and the error occurred during the typesetting stage. The correct version (Fig. 6) of the figure is published in this erratum.

Notes

Declarations

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors’ Affiliations

(1)
Departamento de Farmacología, Facultad de Medicina, Universidad Autónoma de Madrid
(2)
Instituto de Biología Funcional y Genómica, Universidad de Salamanca-CSIC
(3)
Wolfson Institute for Biomedical Research, University College London
(4)
Paul Langerhans-Group, Integrative Physiology, German Diabetes Center
(5)
Department of Pathology, University of Michigan
(6)
Institute of Cancer Sciences, Manchester Cancer Research Centre, University of Manchester

Reference

  1. Peiró C, Romacho T, Azcutia V, Villalobos L, Fernández E, Bolaños JP, Moncada S, Sánchez-Ferrer CF. Inflammation, glucose, and vascular cell damage: the role of the pentose phosphate pathway. Cardiovasc Diabetol. 2016;15:82. doi:https://doi.org/10.1186/s12933-016-0397-2.View ArticlePubMedPubMed CentralGoogle Scholar

Copyright

© The Author(s) 2017

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