Fig. 10From: GLP-1 analog liraglutide-induced cardiac dysfunction due to energetic starvation in heart failure with non-diabetic dilated cardiomyopathyChanged protein expression in cardiac muscles by 10% glucose administration for heart failure deteriorated by high-dose liraglutide. a, b GLUT-4 and CD36 were upregulated further. c, d The levels of the apoptotic marker cleaved caspase-3 and autophagy marker LC3 II/I significantly reduced with 10% glucose administration. n = 6 in each groupBack to article page