Alok Gupta, Pennington Biomedical Research Center, Louisiana State University System, Baton Rouge, LA 70808, USA.
6 October 2010
Table 3 compares the seven obese participants with normal circadian BP variability with the eight obese participants who had abnormalities. The seven obese participants (BMI 32 kg/m2) who had normal circadian BP variability had normal glucose, hs-CRP, fibrinogen, triglycerides, HDL-C and cardiac risk ratios. In contrast the eight obese subjects with abnormal circadian BP variability exhibited (REPLACE majority WITH all) all of the CVD risk parameters outside of the desirable range.
The results from this study show that latent CVD risk in disease-free (healthy) obese adults assessed with no or low risk by conventional risk assessment methods, can be unmasked by simple non-invasive measures. The obese participants exhibiting normal circadian BP variability had normal endothelial function, (REMOVE normotension) normoglycemia and were within the desirable limits for systemic inflammation, triglycerides, HDL-C, and cardiac risk ratios.
In Figure 5, Figure 6, Subjects with No Abnormalities (n=7) (Not n=2).
Erratum:
6 October 2010
Table 3 compares the seven obese participants with normal circadian BP variability with the eight obese participants who had abnormalities. The seven obese participants (BMI 32 kg/m2) who had normal circadian BP variability had normal glucose, hs-CRP, fibrinogen, triglycerides, HDL-C and cardiac risk ratios. In contrast the eight obese subjects with abnormal circadian BP variability exhibited (REPLACE majority WITH all) all of the CVD risk parameters outside of the desirable range.
The results from this study show that latent CVD risk in disease-free (healthy) obese adults assessed with no or low risk by conventional risk assessment methods, can be unmasked by simple non-invasive measures. The obese participants exhibiting normal circadian BP variability had normal endothelial function, (REMOVE normotension) normoglycemia and were within the desirable limits for systemic inflammation, triglycerides, HDL-C, and cardiac risk ratios.
In Figure 5, Figure 6,
Subjects with No Abnormalities (n=7) (Not n=2).
Competing interests
None.