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Table 4 Study cohort of 190 patients suffering from chronic inflammatory bowel diseases.

From: Frequency and significance of the novel single nucleotide missense polymorphism Val109Asp in the human gene encoding omentin in Caucasian patients with type 2 diabetes mellitus or chronic inflammatory bowel diseases

 

Total population

Crohn's Disease

Ulcerative Colitis

n (%)

190 (100%)

128 (67.4%)

62 (32.6%)

age (years) ± SEM

37 ± 10

36 ± 10

39 ± 10

female n (%)

85 (44.7%)

64 (50.0%)

21 (34%)

male n (%)

106 (55.3%)

64 (50.0%)

42 (66%)

Body mass index (BMI) kg/m2 ± SEM

23.8 ± 3.5

23.3 ± 3.6

24.7 ± 3.2

C-reactive protein mg/dl ± SEM

14.6 ± 13.5

15.1 ± 13.0

13.9 ± 14.4

systemic steroids n (%)

79 (41%)

46 (36%)

33 (53%)

topical steroids n (%)

47 (25%)

24 (18%)

23 (37%)

other immuno-suppressants n (%)

48 (25%)

32 (24%)

16 (25%)

Vienna classification [22] n (%): L1

-

21 (16.4%)

-

L2

-

24 (18.8%)

-

L3

-

67 (52.3%)

-

L4

-

16 (12.5%)

-

B1

-

35 (27.3%)

-

B2

-

35 (27.3%)

-

B3

-

58 (45.4%)

-

  1. Vienna classification: L1 = terminal ileum, L2 = colon, L3 = ilecolon, L4 = upper gastrointestinal tract; B1 = non-stricturin, non-penetrating, B2 = stricturing, B3 = penetrating