Figure 5From: High glucose induces human endothelial dysfunction through an Axl-dependent mechanismHG concentrations and Axl transfection in HMEC-1Â s. The Axl transfection significantly reversed HG-induced Akt phosphorylation and VCAM-1 protein expression (A) and increased cell viability and tube formation (B,C) and decreased cell adhesion with monocytes (D).Back to article page