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Ubiquitin–proteasome system; Ischemia–reperfusion; Cardiac dysfunction; Hyperglycemia; Inflammation; Oxidative stress; Autophagy
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doi:10.1186/s12933-015-0258-4
BioMed Central
Cardiovascular Diabetology, doi:10.1186/s12933-015-0258-4
Ubiquitin–proteasome system; Ischemia–reperfusion; Cardiac dysfunction; Hyperglycemia; Inflammation; Oxidative stress; Autophagy
Partial inhibition of the ubiquitin–proteasome system ameliorates cardiac dysfunction following ischemia–reperfusion in the presence of high glucose
Buin Adams
Rudo F Mapanga
M Faadiel Essop
10.1186/s12933-015-0258-4
2010-04-23
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