Fig. 5From: Proteomics of fibrin amyloid microclots in long COVID/post-acute sequelae of COVID-19 (PASC) shows many entrapped pro-inflammatory molecules that may also contribute to a failed fibrinolytic systemA Progression from acute COVID-19 to B long COVID and the persistence of microclots and a general hypoxic state, accompanied by increased circulating antibodies, proteins related to cellular function and liver protein dysfunction (C)Back to article page