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Table 1 Clinical and angiographic characteristics of obstructive-AMI and MINOCA patients

From: Hyperglycemia, inflammatory response and infarct size in obstructive acute myocardial infarction and MINOCA

  Obstructive AMI N = 2450 MINOCA N = 239
STEMI N = 1116 NSTEMI N = 1334   STEMI N = 28 NSTEMI N = 211
Lesion Location    Causes   
 LM lesion, n (%) 47 (4.2) 195 (14.6) Epicardial coronary spasm 1 (3.6) 13 (6.2)
 LAD lesion, n (%) 954 (85.4) 988 (74) SCAD 9 (32.1) 26 (12.3)
 LCx lesion, n (%) 412 (36.9) 520 (38.9) Coronary embolism 4 (14.3) 0 (0)
 RC lesion, n (%) 744 (66.7) 759 (56.9) Atherosclerotic plaque disruption (type I) 5 (17.9) 31 (14.7)
    Supply–demand mismatch (type II) 9 (32.1) 141 (66.8)
Number of Vessels   Number of vessels with stenosis   
 LM, n (%) 37 (3.3) 148 (11.1) LM (1–20%), n (%) 4 (14.3) 1 (0.5)
 1 Vessel, n (%) 667 (59.8) 459 (34.4) 1 Vessel (20–49%), n (%) 3 (10.7) 31 (14.6)
 2 Vessels, n (%) 303 (27.2) 383 (28.7) 2 Vessels (20–49%), n (%) 3 (10.7) 13 (6.2)
 3 Vessels, n (%) 109 (9.7) 344 (25.8) 3 Vessels (20–49%), n (%) 0 (0) 3 (1.4)
  1. AMI Acute Myocardial Infarction, LAD Left Anterior Descending artery, LCx Left Circumflex, LM Left Main, MINOCA myocardial Infarction with Non-Obstructive Coronary Arteries, NSTEMI Non-ST-segment elevation myocardial infarction, RC Right Coronary artery, STEMI ST-segment elevation myocardial infarction, SCAD Spontaneous coronary artery dissection