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Table 1 Clinical and angiographic characteristics of obstructive-AMI and MINOCA patients

From: Hyperglycemia, inflammatory response and infarct size in obstructive acute myocardial infarction and MINOCA

 

Obstructive AMI N = 2450

MINOCA N = 239

STEMI N = 1116

NSTEMI N = 1334

 

STEMI N = 28

NSTEMI N = 211

Lesion Location

  

Causes

  

 LM lesion, n (%)

47 (4.2)

195 (14.6)

Epicardial coronary spasm

1 (3.6)

13 (6.2)

 LAD lesion, n (%)

954 (85.4)

988 (74)

SCAD

9 (32.1)

26 (12.3)

 LCx lesion, n (%)

412 (36.9)

520 (38.9)

Coronary embolism

4 (14.3)

0 (0)

 RC lesion, n (%)

744 (66.7)

759 (56.9)

Atherosclerotic plaque disruption (type I)

5 (17.9)

31 (14.7)

   

Supply–demand mismatch (type II)

9 (32.1)

141 (66.8)

Number of Vessels

 

Number of vessels with stenosis

  

 LM, n (%)

37 (3.3)

148 (11.1)

LM (1–20%), n (%)

4 (14.3)

1 (0.5)

 1 Vessel, n (%)

667 (59.8)

459 (34.4)

1 Vessel (20–49%), n (%)

3 (10.7)

31 (14.6)

 2 Vessels, n (%)

303 (27.2)

383 (28.7)

2 Vessels (20–49%), n (%)

3 (10.7)

13 (6.2)

 3 Vessels, n (%)

109 (9.7)

344 (25.8)

3 Vessels (20–49%), n (%)

0 (0)

3 (1.4)

  1. AMI Acute Myocardial Infarction, LAD Left Anterior Descending artery, LCx Left Circumflex, LM Left Main, MINOCA myocardial Infarction with Non-Obstructive Coronary Arteries, NSTEMI Non-ST-segment elevation myocardial infarction, RC Right Coronary artery, STEMI ST-segment elevation myocardial infarction, SCAD Spontaneous coronary artery dissection