Fig. 7

mAb A treatment inhibits AMP-activated protein kinase (AMPK) and acetyl CoA carboxylase (ACC) phosphorylation and increases malonyl CoA decarboxylase MCD expression post-MI. a Representative blots for fatty acid regulatory signalling. a Densiometric analysis for b phosphorylated 5′ AMP-activated protein kinase (p-AMPK^Tyr172)/AMPK, c phosphorylated acetyl CoA carboxylase (p-ACC^Ser79)/ACC, d malonyl CoA decarboxylase (MCD)/α-tubulin. Protein bands were normalised for either to their correspondent total protein band or to α-tubulin bands for intra-experiment variation. *p < 0.05. n = 6/group. S sham, V vehicle, Ab mAb A, MI myocardial infarction