Donor artery stenosis interactions with diastolic blood pressure on coronary collateral flow in type 2 diabetic patients with chronic total occlusion

Table 2 Aortic blood pressure in relation to coronary flow index in diabetic and non-diabetic patients according to stenosis of PCDA

Aortic BP	Mild					Moderate					Severe
	Diabetes (n = 99)		Non-diabetes (n = 132)		Pint	Diabetes (n = 75)		Non-diabetes (n = 60)		Pint	Diabetes (n = 46)		Non-diabetes (n = 28)		Pint
	β ± SE	P	β ± SE	P	Pint	β ± SE	P	β ± SE	P	Pint	β ± SE	P	β ± SE	P	Pint
Systolic	− 0.284 ± 0.073	< 0.001	− 0.352 ± 0.070	< 0.001	0.325	− 0.013 ± 0.062	0.840	0.065 ± 0.158	0.682	0.239	0.154 ± 0.204	0.464	0.025 ± 0.085	0.766	0.651
Diastolic	− 0.275 ± 0.079*	0.001	− 0.525 ± 0.065	< 0.001	0.168	− 0.019 ± 0.066^#	0.774	− 0.303 ± 0.114	0.011	0.008	0.317 ± 0.087	0.001	0.199 ± 0.183	0.296	0.032
Mean	− 0.334 ± 0.074^&	< 0.001	− 0.540 ± 0.061	< 0.001	0.081	− 0.018 ± 0.063	0.779	− 0.158 ± 0.136	0.253	0.067	0.604 ± 0.219^&	0.016	0.147 ± 0.087	0.008	0.669

Values are regression coefficient (β) ± SE, derived from multiple linear regression analyses of coronary flow index with central aortic systolic, diastolic and mean blood pressure, respectively, after adjustment for gender, age, body mass index, current smoking, history of hypertension and dyslipidemia, prior myocardial infarction, severity of coronary artery disease, GFR, total and HDL cholesterol ratio, log-transferred hsCRP and left ventricular ejection fraction. P values for interaction in group of mild, intermediate and severe stenosis of PCDA are given
GFR glomerular filtration rate, HDL high-density lipoprotein, hsCRP high-sensitivity C reactive protein, PCDA predominant collateral donor artery
* P < 0.001, ^# P < 0.01 and ^& P < 0.05 vs. the corresponding β value in non-diabetes

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