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Table 1 Baseline characteristics of study cohort

From: Insulin resistance, endothelial function, angiogenic factors and clinical outcome in non-diabetic patients with chest pain without myocardial perfusion defects

 

Whole study population (n = 365)

Without perfusion defects (n = 238)

Age (years)

62 ± 9

61 ± 9

Women

202 (55 %)

150 (63 %)

Body mass index

25.7 ± 3.5

25.4 ± 3.5

Current smoker

47 (13 %)

31 (13 %)

Family history of CAD

142 (39 %)

89 (37 %)

ACE-inhibitors

69 (19 %)

32 (13 %)

Beta-blockers

173 (47 %)

96 (40 %)

Statins

146 (40 %)

83 (35 %)

Aspirin

178 (49 %)

102 (43 %)

Systolic blood pressure (mmHg)

144 ± 23

144 ± 22

Known CAD

94 (26 %)

44 (18 %)

Previous MI

51 (14 %)

19 (8 %)

Triglycerides (mmol/L) (n = 362)

1.2 (0.8–1.6)

1.1 (0.8–1.3) (n = 235)

Cholesterol (mmol/L) (n = 360)

5.4 ± 1.3

5.4 ± 1.1 (n = 234)

HDL (mmol/L)

1.47 (1.24–1.72)

1.49 (1.29–1.75)

ApoB/ApoA1

0.64 (0.53–0.79)

0.62 (0.52–0.76)

Fasting glucose (mmol/L)

5.3 ± 0.5

5.3 ± 0.5

Insulin (pmol/L)

94 (69–125)

92 (69–122)

HbA1c (mmol/mol, %)

36.6 ± 3.8 (5.5 ± 0.3 %)

36.2 ± 3.5 (5.5 ± 0.32 %)

HOMA-IR

3.1 (2.3–4.3)

3.1 (2.3–4.1)

RHI (n = 345)

1.89 (1.55–2.47)

1.91 (1.59–2.48) (n = 225)

  1. Values are displayed as mean ± SD or median and interquartile range for continuous variables and frequency and percentages for categorical variables
  2. ApoA apolipoprotein A, ApoB apolipoprotein B, HbA 1c glycosylated hemoglobin, HDL high density lipoprotein cholesterol. MI myocardial infarction, Known CAD previously known coronary artery disease, RHI reactive hyperemic index