Fig. 5From: Urinary alpha-1 antitrypsin and CD59 glycoprotein predict albuminuria development in hypertensive patients under chronic renin-angiotensin system suppressionMain alterations in response to albuminuria progression point to inflammation and coagulation disorders as main biological pathways involved. Differences observed already in normoalbuminuria condition reveal subjacent mechanisms associated with cardiovascular risk taking place already in asymptomatic stagesBack to article page