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Figure 5 | Cardiovascular Diabetology

Figure 5

From: Chronic Rho-kinase inhibition improves left ventricular contractile dysfunction in early type-1 diabetes by increasing myosin cross-bridge extension

Figure 5

Diastolic and peak systolic myosin mass transfer. Diastolic (a) and peak systolic (b) myosin mass transfer in the epicardium, subepicardium and subendocardium of the left ventricle. Diastolic myosin mass transfer did not significantly differ between vehicle-treated control and diabetic rats or fasudil-treated (10 mg/kg/day) control and diabetic rats in the epicardial layer. In the subepicardial and subendocardial layers, no significant changes were observed in diastolic myosin mass transfer in diabetic rats compared to controls, or to fasudil-treated diabetic rats. Peak systolic myosin mass transfer was similar in all LV layers in diabetic rats compared to controls. Fasudil treatment of diabetic rats did not change subendocardial systolic myosin mass transfer. Data expressed as mean ± SEM. N = 4–6 per group.

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