Fig. 5From: Tissue inflammation and nitric oxide-mediated alterations in cardiovascular function are major determinants of endotoxin-induced insulin resistanceTissue blood flow (ml · mg−1 · min−1) for muscle, liver, intestines in saline (SAL) or sodium nitroprusside (NO)-treated wild-type (WT) mice (a) (Group 1C). Kidney blood flow was measured as clearance (ml · kg−1 · min−1) of para-aminohippuric acid (PAH) clearance (b). Data are mean ± SEM (n = 3–5). *p < 0.05 vs. WT + SAL compared by t-test. NS not significant (p ≥ 0.05)Back to article page