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Table 4 Biometric, lipoprotein profile distributions, assessment-insulin resistance and blood glucose distributions in relation to the PPARγ C161→T genotypes in CAD patients

From: PPARγ gene C161T substitution alters lipid profile in Chinese patients with coronary artery disease and type 2 diabetes mellitus

  PPARγ genotypes
Groups CC (n = 50) CT (n = 25) TT (n = 3) CT+TT (n = 28)
Age 62.40 ± 12.04 67.75 ± 11.18 63.00 ± 8.73 66.80 ± 10.12
Weight (kg) 62.93 ± 9.59 58.23 ± 7.13 58.67 ± 7.51 58.32 ± 6.9 2
Height (m) 1.61 ± 0.07 1.63 ± 0.09 1.61 ± 0.06 1.63 ± 0.08
BMI (kg/m2) 22.48 ± 6.60 21.79 ± 3.14 23.01 ± 3.14 22.05 ± 3.06
TC (mmol/L) 4.37 ± 1.32 4.76 ± 1.36 3.82 ± 0.88 4.57 ± 1.31
Trig (mmol/L) 1.65 ± 0.89 1.51 ± 0.85 1.70 ± 0.82 1.55 ± 0.82
HDL (mmol/L) 1.21 ± 0.23 1.58 ± 0.62 1.13 ± 0.18 1.49 ± 0.59
LDL (mmol/L) 2.82 ± 0.69 2.77 ± 1.05 2.09 ± 0.60 2.63 ± 0.99
apoA1 (g/L) 1.29 ± 0.17 1.39 ± 0.22 1.27 ± 0.08 1.37 ± 0.20
apoB (g/L) 1.08 ± 0.11 0.87 ± 0.29 0.74 ± 0.28 0.85 ± 0.29
Lp(a) (mg/L) Δ 1.90 ± 0.64 1.89 ± 0.58 1.25 ± 0.03 1.78 ± 0.58
FBS (mmol/L) 5.49 ± 0.77 5.78 ± 0.75 5.46 ± 1.06 5.71 ± 0.79
PPBS (mmol/L) 6.38 ± 1.22 8.17 ± 2.03 6.87 ± 0.28 7.87 ± 1.85
RI Δ 1.08 ± 0.16 1.03 ± 0.21 1.12 ± 0.14 1.05 ± 0.19
  1. No statistically significant difference was observed between the gene type groups (P > 0.05)
  2. Δ The measurement material were descriptive statistics of skewed distribution, and counted after logarithm