Figure 3From: High glucose induces human endothelial dysfunction through an Axl-dependent mechanismHG concentrations and HMEC-1 cell viability, adhesion, tubule formation and migration. (A,B) HG significantly decreased HMEC-1Â s survival and tube formation with dose-dependent manner. (C,D) HG promoted significant monocyte-EC adhesion but did not influence HMEC-1 cell migration.Back to article page